Aryl Organophosphate Esters and Hemostatic Disruption: Identifying Risk through Machine Learning and Experimental Validation.

Journal: Environmental science & technology
Published Date:

Abstract

Organophosphate esters (OPEs) have emerged as a significant environmental concern due to their widespread occurrence and potential human health risks. The presence of OPEs in human blood suggests direct interactions with hematological components, which may compromise hemostatic balance and lead to adverse health outcomes. Despite the critical role of hemostatic balance in maintaining blood stability, the effects of OPEs on this system remain poorly understood. This investigation was undertaken to delineate the effects and potential mechanisms of OPEs that modulate hemostasis, utilizing approach and high-throughput investigation. We analyzed 85 environmentally prevalent OPEs for their structural descriptors and affinity for proteins essential to hemostatic function. The multiple linear regression implicated aryl-OPEs, distinguished by their benzene ring scaffold, as potent disruptors of hemostatic balance. This analysis result was rigorously validated through the hemostatic balance assays. Further investigation through network toxicology, artificial intelligence (α-Fold) algorithms, and an agonist cotreatment assay revealed proliferator-activated receptor γ (PPARγ) as a key mediator of aryl-OPEs induced hemostatic disruption. By integrating experimental insights with exposure data, we concluded that specific aryl-OPEs, such as bisphenol a bis (diphenyl phosphate) (BDP) and cresyl diphenyl phosphate (CDP), pose a moderate risk to the hemostatic balance of the general population. Our findings not only contribute to the prioritization of OPEs risk management but also establish a methodology for assessing the hematological toxicity of emerging pollutants.

Authors

  • Pu Chen
    Department of Biomedical Engineering, Wuhan University School of Basic Medical Sciences, Wuhan, 430071, China.
  • Ziyuan Li
    Department of Radiology, Peking University First Hospital, No. 8, Xishiku Street, Xicheng District, Beijing, 100034, China.
  • Gan Miao
    Department of Occupational and Environmental Health, School of Public Health, Qingdao University, Qingdao 266071, China.
  • Xiaopeng Tang
    Department of Biochemistry and Molecular Biology, School of Basic Medicine, Qingdao University, Qingdao 266071, China.
  • Chengying Zhou
    Department of Occupational and Environmental Health, School of Public Health, Qingdao University, Qingdao 266071, China.
  • Li Zhao
    International Initiative on Spatial Lifecourse Epidemiology (ISLE), the Netherlands; Department of Health Policy and Management, West China School of Public Health/West China Fourth Hospital, Sichuan University, Chengdu, Sichuan, 610041, China; Research Center for Healthy City Development, Sichuan University, Chengdu, Sichuan, 610041, China; Healthy Food Evaluation Research Center, Sichuan University, Chengdu, Sichuan, 610041, China.
  • Xiaoting Jin
    Department of Occupational and Environmental Health, School of Public Health, Qingdao University, Qingdao 266071, China.
  • Guangbo Qu
    State Key Laboratory of Environmental Chemistry and Ecotoxicology, Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences, Beijing 100085, P. R. China.
  • Yuxin Zheng
    Department of Occupational and Environmental Health, School of Public Health, Qingdao University, Qingdao 266071, China.
  • Guibin Jiang
    State Key Laboratory of Environmental Chemistry and Ecotoxicology, Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences, Beijing 100085, China.