Serum Peroxisome Proliferator-activated Receptor Gamma Coactivator-1α Related to Myocardial Energy Expenditure in Patients With Chronic Heart Failure.

Journal: The American journal of the medical sciences
Published Date: Dec 7, 2018

Abstract

BACKGROUND: Peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α) plays key roles in controlling cardiac metabolism and function. Myocardial energy expenditure (MEE) can reflect myocardial energy metabolism and cardiac function. Whether the variation of PGC-1α can influence MEE levels in chronic heart failure (CHF) is unclear. Therefore, we investigated the relationship between PGC-1α and MEE.

Authors

  • Pingan Chen
    State Key Laboratory of Organ Failure Research, Department of Cardiology and; Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China; Key Laboratory For Organ Failure Research, Ministry of Education of the People's Republic of China, Guangzhou, Guangdong, China; Department of Cardiology and.
  • Qiong Zhan
    State Key Laboratory of Organ Failure Research, Department of Cardiology and; Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China; Key Laboratory For Organ Failure Research, Ministry of Education of the People's Republic of China, Guangzhou, Guangdong, China.
  • Yujia Bai
    Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China.
  • Xingfu Huang
    Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China.
  • Peng Wang
    Neuroengineering Laboratory, School of Biomedical Engineering and Technology, Tianjin Medical University, Tianjin, China.
  • Yizhi Pan
    Department of Cardiology and.
  • Shaonan Li
    Department of Cardiology and.
  • Shenshen Fu
    Ultrasonic Department, Guangzhou First People's Hospital, Guangzhou Medical University, Guangzhou, Guangdong, China.
  • Wenyan Lai
    State Key Laboratory of Organ Failure Research, Department of Cardiology and; Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China; Key Laboratory For Organ Failure Research, Ministry of Education of the People's Republic of China, Guangzhou, Guangdong, China.
  • Qingchun Zeng
    State Key Laboratory of Organ Failure Research, Department of Cardiology and; Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China; Key Laboratory For Organ Failure Research, Ministry of Education of the People's Republic of China, Guangzhou, Guangdong, China.
  • Hao Ren
    Department of Rheumatology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China. Electronic address: renhao67@aliyun.com.
  • Dingli Xu
    State Key Laboratory of Organ Failure Research, Department of Cardiology and; Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong, China; Key Laboratory For Organ Failure Research, Ministry of Education of the People's Republic of China, Guangzhou, Guangdong, China. Electronic address: dinglixu@fimmu.com.

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