Histone deacetylase 2 is decreased in peripheral blood pro-inflammatory CD8+ T and NKT-like lymphocytes following lung transplant.

Journal: Respirology (Carlton, Vic.)
Published Date:

Abstract

BACKGROUND AND OBJECTIVE: Immunosuppression therapy following lung transplantation fails to prevent chronic rejection in many patients, which is associated with lack of suppression of cytotoxic mediators and pro-inflammatory cytokines in peripheral blood T and natural killer T (NKT)-like cells. Histone acetyltransferases (HATs) and histone deacetylases (HDACs) upregulate/downregulate pro-inflammatory gene expression, respectively; however, differences in the activity of these enzymes following lung transplant are unknown. We hypothesized decreased HDAC2 expression and increased HAT expression in pro-inflammatory lymphocytes following lung transplant.

Authors

  • Greg Hodge
    Lung Research, Hanson Institute, Adelaide, South Australia, Australia.
  • Sandra Hodge
    Lung Research, Hanson Institute, Adelaide, South Australia, Australia.
  • Chien-Li Holmes-Liew
    Lung Research, Hanson Institute, Adelaide, South Australia, Australia.
  • Paul N Reynolds
    Lung Research, Hanson Institute, Adelaide, South Australia, Australia.
  • Mark Holmes
    Lung Research, Hanson Institute, Adelaide, South Australia, Australia.