Inflammatory markers modify the risk of recurrent coronary events associated with apolipoprotein A-I in postinfarction patients.

Journal: Journal of clinical lipidology
PMID: 28391888

Abstract

BACKGROUND: Laboratory findings have suggested that systemic and vascular inflammation can impair the antiatherogenic function of high-density lipoproteins (HDLs). However, evidence from population studies is sparse.

Authors

  • Meng Wang
    State Key Laboratory of Urban Water Resource and Environment, School of Environment, Harbin Institute of Technology, Harbin 150001, China.
  • James Corsetti
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY, USA.
  • Scott McNitt
    Heart Research Follow-up Program, University of Rochester Medical Center, Rochester, NY, USA.
  • David Q Rich
    Division of Epidemiology, Department of Public Health Sciences, University of Rochester Medical Center, Rochester, NY, USA.
  • Charles E Sparks
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY, USA.
  • Arthur J Moss
    Heart Research Follow-up Program, University of Rochester Medical Center, Rochester, NY, USA.
  • Wojciech Zareba
    Heart Research Follow-up Program, University of Rochester Medical Center, Rochester, NY, USA. Electronic address: wojciech_zareba@urmc.rochester.edu.

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