Cortical frequency-specific plasticity is independently induced by intracortical circuitry.

Journal: Neuroscience letters
PMID:

Abstract

Auditory learning induces frequency-specific plasticity in the auditory cortex. Both the auditory cortex and thalamus are involved in the cortical plasticity; however, the precise role of the intracortical circuity remains unclear until the contributions of the thalamocortical inputs are controlled. Here, we induced cortical plasticity by local activation of the primary auditory cortex (AI) via intracortical electrical stimulation (ES) in C57 mice and found a similar pattern of cortical plasticity was induced by ES when the auditory thalamus was inactivated or remained active during the ES. The best frequencies (BFs) of the recorded cortical neurons shifted towards the BFs of the electrically stimulated ones. In addition, the BF shifts were linearly correlated to the BF differences between the recorded and stimulated cortical neurons. More importantly, the ratio of the linear function with thalamic inactivation was nearly the same as the ratio of the linear function in the control condition. Our data show that cortical frequency-specific plasticity was induced by ES with or without the thalamic inactivation; thus intracortical circuitry can be independently responsible for cortical frequency-specific plasticity.

Authors

  • Lingzhi Kong
    Department of Physiology and Pharmacology, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, Alberta, T2N 4N1, Canada; Allied Health School, Beijing Language and Culture University, Beijing, 100871, China.
  • Shaohui Wang
    Hubei Key Lab of Genetic Regulation and Integrative Biology, School of Life Sciences, Central China Normal University, Wuhan, 430079, China.
  • Xiuping Liu
    Department of Physiology and Pharmacology, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada; College of Nursing, Hebei University, Baoding, Hebei, China.
  • Liang Li
    School of Psychological and Cognitive Sciences, Peking University, Beijing, 100871, China.
  • Michael Zeeman
    Department of Physiology and Pharmacology, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, Alberta, T2N 4N1, Canada.
  • Jun Yan
    Department of Statistics, University of Connecticut, Storrs, CT 06269, USA.