Development of non-bias phenotypic drug screening for cardiomyocyte hypertrophy by image segmentation using deep learning.

Journal: Biochemical and biophysical research communications
Published Date:

Abstract

The number of patients with heart failure and related deaths is rapidly increasing worldwide, making it a major problem. Cardiac hypertrophy is a crucial preliminary step in heart failure, but its treatment has not yet been fully successful. In this study, we established a system to evaluate cardiomyocyte hypertrophy using a deep learning-based high-throughput screening system and identified drugs that inhibit it. First, primary cultured cardiomyocytes from neonatal rats were stimulated by both angiotensin II and endothelin-1, and cellular images were captured using a phase-contrast microscope. Subsequently, we used a deep learning model for instance segmentation and established a system to automatically and unbiasedly evaluate the cardiomyocyte size and perimeter. Using this system, we screened 100 FDA-approved drugs library and identified 12 drugs that inhibited cardiomyocyte hypertrophy. We focused on ezetimibe, a cholesterol absorption inhibitor, that inhibited cardiomyocyte hypertrophy in a dose-dependent manner in vitro. Additionally, ezetimibe improved the cardiac dysfunction induced by pressure overload in mice. These results suggest that the deep learning-based system is useful for the evaluation of cardiomyocyte hypertrophy and drug screening, leading to the development of new treatments for heart failure.

Authors

  • Jin Komuro
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo, 160-8582, Japan.
  • Yuta Tokuoka
    Department of Biosciences and Informatics, Keio University, Yokohama-shi, Kanagawa, Japan.
  • Tomohisa Seki
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan; Department of Emergency and Critical Care Medicine, Keio University School of Medicine, Tokyo 160-8582, Japan.
  • Dai Kusumoto
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan; Department of Emergency and Critical Care Medicine, Keio University School of Medicine, Tokyo 160-8582, Japan.
  • Hisayuki Hashimoto
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo, 160-8582, Japan.
  • Toshiomi Katsuki
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan.
  • Takahiro Nakamura
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo, 160-8582, Japan. Electronic address: ta-nakamura11@keio.jp.
  • Yohei Akiba
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo, 160-8582, Japan. Electronic address: yohey0814@gmail.com.
  • Thukaa Kuoka
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo, 160-8582, Japan. Electronic address: thukaa.kuoka@gmail.com.
  • Mai Kimura
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan.
  • Takahiro Yamada
    Division of Positron Emission Tomography, Institute of Advanced Clinical Medicine, Kindai University, Osaka, Japan.
  • Keiichi Fukuda
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan.
  • Akira Funahashi
    Department of Biosciences and Informatics, Keio University, Kanagawa, Japan.
  • Shinsuke Yuasa
    Department of Cardiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan. Electronic address: yuasa@keio.jp.